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dc.coverage.spatialValparaíso
dc.creatorRetamal-Díaz, Angello Ricardo
dc.creatorTognarelli-Torres, Eduardo Ignacio
dc.creatorÁlvarez, Diana C M
dc.creatorKalergis, Alexis M
dc.creatorGonzález-Muñoz, Pablo Alberto
dc.date.accessioned2019-03-27T15:56:45Z
dc.date.available2019-03-27T15:56:45Z
dc.date.issued2017
dc.identifier.urihttp://hdl.handle.net/10533/233586
dc.description.abstractHerpes simplex virus 2 (HSV-2) is the primary cause of genital ulcers and neonatal encephalitis. Noteworthy, HSV-2 interferes with the maturation and viability of dendritic cells (DCs), likely affecting the capacity of the host to mount effective adaptive
dc.language.isospa
dc.titleA herpes simplex virus type 2 deleted for glycoprotein D enables dendritic cells to activate CD4+ and CD8+ T-cells
dc.typePonencia
dc.identifier.folio1140011
dc.country.isoChile
dc.description.conicytprogramFONDECYT
dc.relation.ncongress
dc.relation.projectidinfo:eu-repo/grantAgreement/Fondecyt/1140011
dc.rights.driverinfo:eu-repo/semantics/openAccess
dc.date.start2017
dc.date.end2017
dc.description.shortconicytprogramFONDECYT
dc.relation.namecongReunión Anual de la Asociación Chilena de Inmunología - ASOCHIN


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